By William T. Cefalu

The 1st atlas committed particularly to cardio-metabolic danger, this ebook will offer a concise visible primer at the pathophysiology, epidemiology, prognosis, remedy, and scientific and radiologic positive aspects of this sickness. Describing contemporary care ideas and present practices in sufferer overview, this resource will let clinicians to competently determine and deal with these sufferers at elevated threat for metabolic syndrome, heart problems, and diabetes.

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Thus, an individual with obesity and insulin resistance, depending on the stage of compensation for the insulin resistance, may have euglycemia, impaired fasting glucose, impaired glucose tolerance, or overt hyperglycemia confirming the diagnosis of type 2 diabetes. 3 Electron micrograph of an islet of Langerhans from a normal pancreas showing mainly insulin storage granules in a pancreatic β-cell. A larger α (glucagon) cell is also seen. The normal adult pancreas contains around one million islets comprising mainly β-cells (producing insulin), α-cells (glucagon), D cells (somatostatin), and PP (pancreatic polypeptide) cells.

Despite similar body mass index (BMI) measurements for the male (a), and female (b) subjects, there are significant differences in abdominal fat distribution. BW, body weight; VF, visceral fat; SF, superficial fat; DSF, deep subcutaneous fat; SSF, superficial subcutaneous fat. 11 Relationship between visceral adipose tissue and skeletal muscle insulin action in both men and women. LBM, lean body mass. 12 Relationship between aging and accumulation of visceral fat. As shown in left panel, this study evaluated both men and women through seven decades.

J Clin Endocrinol Metab 2004; 89: 2548–56 9. Ravussin E, Smith SR. Increased fat intake, impaired fat oxidation, and failure of fat cell proliferation result in ectopic fat storage, insulin resistance, and type 2 diabetes mellitus. Ann NY Acad Sci 2002; 967: 363–78 10. Weyer C, Foley JE, Bogardus C, et al. Enlrged subcutaneious abdominal adipocyte sixe, but not obesity itself, predicts type II diabetes independent of insulin resistance. Diabetologia 2000; 43: 1498–506 11. Despres JP, Lemieux I, Proud‘homme D.

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Atlas of Cardiometabolic Risk by William T. Cefalu
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